Cancer comes from a simple fungal infection and It can be cured

My idea is that cancer doesn't depend on mysterious causes (genetic, immunological or auto immunological as the official oncology proposes, but it comes down from a simple fungal infection, whose destroying power in the deep tissues is actually under estimated.

Premise

The present work is based on the conviction, supported by many years of observations, comparisons and experiences, that the necessary and sufficient cause of the tumour is to be sought in the vast world of the fungi, the most adaptable, aggressive and evolved micro-organisms known in nature.
I have tried many times to explain this theory to leading institutions involved in cancer issues (the Ministry of Health, the Italian Medical Oncological Association, etc.) elaborating on my thinking, but I have been brushed aside because of the impossibility of setting my idea in a conventional context.
A different, international audience represents the possibility of sharing a view about health, which differs, from what is widely accepted by today's medical community, either officially or from the sidelines.

There is an opposition between the allopathic and the Hippocratic medical ideal. The former has the disadvantage of its inability to consider the individual as a whole. Therefore it brings with it all the distortions and aberrations which such a point of view entails (excessive specialisation, therapeutic aggressiveness, superficiality, harmfulness etc.). The latter approach instead tends in the direction of being too generic, non-scientific, and devoid of therapeutic incisiveness.
The position that I promote represents instead a meeting point of these two conceptions of health, since, from the conceptual point of view, it sublimates and adds value to both, while highlighting how they both are victims of a common conformist language.
The hypothesis of a fungal aetiology in chronic-degenerative illness, able to connect the ethical qualities of the individual with the development of specific pathologies, reconciles the two orientations (allopathic and holistic) of medicine. The hypothesis is a strong candidate for being that missing element of psychosomatics that has been sought but never found by one of the fathers of psychosomatics, Wiktor Von Weiszcker.

In considering the biological dimensions of the fungi, for instance, it is possible to compare the different degrees of pathogenicity in relation to the condition of organs, tissues and cells of a guest organism, which in turn also and especially depend on the behaviour of the individual.
Each time the recuperative abilities of a known psycho-physic structure are exceeded, there is an inevitable exposure, even considering possible accidental cofounders, to the aggression -- even at the smallest dimensions -- of those external agents that otherwise would be harmless.
In the presence of an indubitable connection between patient morale and disease it is no longer legitimate to separate the two domains (allopathic and naturopathic) which are both indispensable for improving the health of individuals.
The Platonic separation of the human mind from the human body, responsible for the present mechanistic and materialistic character of today's medicine, is outdated. So is the pessimistic Kantian position concerning integration of the rational and emotional sides of man ("the starred sky above me, the moral law within me"), which generates the present myopia of today's medical epistemology. With such outdated cognitive frameworks inevitably come all the mindsets that carry similar restrictive and limiting presuppositions.

Candida Albicans: Necessary and Sufficient Cause of Cancer

When facing the most pressing contemporary medical problem, cancer, the first thing to do is to admit that we still do not know its real cause. However treated in different ways by both official and alternative medicine, an aural of mystery still exists around its real generative process.
The attempt to overcome the present impasse must therefore and necessarily go through two separate phases: a critical one that exposes the present limitations of oncology, and a constructive one capable of proposing a therapeutic system based on a new theoretical point of departure.
In agreement with the most recent formulation of scientific philosophy, which suggests a counter-inductive approach where it is impossible to find a solution with the conceptual tools that are commonly accepted, only one logical formulation emerges; that is, to refuse the oncological principle which assumes cancer is generated by acellular reproductive anomaly.
However, if the fundamental hypoth esis of cellular reproductive anomaly is questioned, it becomes clear that all the theories based on this hypothesis are inevitably flawed.
It follows that both an auto-immunological process, in which the body's defence mechanisms against external agents turn their destructive capacity against internal constituents of the body, and an anomaly of the genetic structure implicated in the development of auto-destruction, are inevitably disqualified.

Moreover, the common attempt to construct theories about multiple causes that have an oncogenic effect on cellular reproduction sometimes seems like a concealing screen, behind which there is nothing but a wall. These theories propose endless causes that are more or less associated with each other; and this means in reality that no valid causes are found. The invocation in turn of smoking, alcohol, toxic substances, diet, stress, psychological factors, etc., without a properly defined context, causes confusion and resignation, and creates even more mystification around a disease which may turn out to be simpler than it is depicted to be.
As background information, it is important to review the picture of presumed genetic influences in the development of cancer processes as they are depicted by molecular biologists. These are the scientists who perform research on infinitesimally small cellular mechanisms, but who in real life never see a patient. All present medical s ystems are based on this research, and thus, unfortunately, all therapies currently performed.
The main hypothesis of a genetic neoplastic causality is essentially reduced to the fact that the structures and the mechanism in charge of normal reproductive cellular activity become, for undefined causes, capable of an autonomous behaviour that is disjointed from the overall tissular economy.

The genes that normally have a positive role in cellular reproduction are, then, imprecisely referred to as proto-oncogenes; those which inhibit cellular reproduction are called suppressor genes or recessive oncogenes.
Both endogenous (never demonstrated) and exogenous cellular factors -- that is, those carcinogenic elements that are usually invoked -- are held responsible for the neoplastic degeneration of the tissues.
In J.H. Stein (Medicina Interna - Internal Medicine, Mosby Year Book inc.1994, St. Louis, Missouri, 4th edition, Milano, 1995, page 1186 -1187) the following is reported:
The mitogenic signals, from the microenvironment or from more distant areas of influence, are transmitted to the cells through numerous receptive structures that are associated to the plasmatic membrane.
Among these structures, the ones that have been studied most exhaustively are receptors with an external domain for the binder, a transmembranic domain and a cytoplasma tic domain with a thyrosinkinase activity.

Besides these, it is thought that at least seven distinct classes of molecules participate in the transmission of the mutagenic signal:
1) receptors coupled to G proteins
2) ionic channels
3) receptors with intrinsic activity guanil cyclase
4) receptors for many lymphofokines, cytokines and growth factors (interleukine, eritropoietine, etc.)
5) receptors for the phosphothyrosine phosphorilase activity
6) nuclear receptors belonging to the supergenic family of the receptor for steroidal estrogenic and thyroidal hormones
7) Finally, increasing numbers of tests suggest that the adhesion molecules expressed on the surface of the cells communicate with the microenvironment in ways that produce very important consequences for cellular growth and differentiation.

From a very superficial analysis of this presumed oncological picture, however, it seems to be clear how the assertion of all this unstoppable genetic hyperactivity, generated by elements that almost seem to lurk in the realms of the sinister and the monstrous, and that therefore suggest the existence of God-knows-what abysmal mechanisms that can only be deciphered with equally abysmal conceptual mechanisms -- all this can do nothing more that unveil the abysmal stupidity that is at the basis of this way of conceiving things.
What is even more serious is the fact that nobody in the present health establishment seems to question the above-mentioned stupidities. All those who work in the field do nothing but repeat the stale litany of reproductive cellular anomalies on a genetic basis.
Since in this state of affairs the present medical theory shows an impoverishment and a superficiality that are indeed abysmal, it is better to look for new horizons and conceptual i nstruments that are capable of unearthing a real and unique neoplastic aetiology.
After so many years of failure and suffering it is time to rejuvenate minds with new and productive juices. Arguments for mysterious and complex genetic factors, a monstrous reproductive capacity by a pathologic entity capable of tearing apart any tissue, the idea that there is an implicit and ancestral tendency of the human organism to deviate in an auto-destructive sense -- these and other similar arguments, spiced with exponentially multiplying numbers of "ifs" and "maybes" -- it all has the flavour more of raving free-association than of a healthy scientific discourse.

Once the present oncological perspectives have been refuted, however, it is legitimate to ask how the successes achieved by official medicine and by alternative medicine have to be classified.
To this end, it is useful to remember that contemporary epistemology has demonstrated how the contributions to causality of contextual and co-textual elements of a theory, if they cannot be defined, are therefore chancy, especially in ultra-dimensional space, that is, in the microscopic dimension.

In practical terms, this means that data or positive facts that are considered proof when concerning a basic principle (for example, the above-mentioned cellular reproductive anomalies), and therefore obtained by utilising a limited number of variables next to the complexity of human disease, cannot be trusted, since they work only from the initial hypothetical functions.
Where, in fact, we admit the possibility of improvements or cures, it is not admissible from the logical point of view attribute them to this or that method of cure that is more of less official, since it is not possible to specify and include all or the majority of the components that are at play in theobjectman, in whom conditions of certainty cannot exist.
Paradoxically, the possible positive effect of each therapeutic system could derive from elements that are not foreseen and are unknown to all. Those elements, however, could be influenced by or determined to some extent by one another.
We may find ourselves in a position in which everybody rightfully has the right to promote his point of view, without knowing the real reasons for his successes.
In this case, then, even the most rigorous experimentation takes on a fictional character rather than the function of a true correspondence with reality, and the end result is a continuous sterilepetitio principii.

If we then put aside completely the conceptual frame of contemporary oncology with all its interpretative variables of genetic, immuncological and toxicological character, what is left as the only logical, practicable way is the domain of the infectious diseases, to be seen and reconsidered with different eyes that has been the case so far.
Two considerations support such a conclusion. One is of a historical nature, and the other is of an epidemiological nature. The former derives from the fact that, in the therapeutical approach to the patient, the improvement in quality, that is the possibility of a real cure for the patient, has been determined almost exclusively by the development of microbiology. The latter derives from the analysis of life expectancy that has taken place in the last decades which, since it is associated with an inevitable change of the sthenicity of individuals, can be hypothesised as a determining factor in the development atypical infectious p athologies.
In order to find the possible carcinogenicens morbion the horizon of microbiology, it appears useful to return to the basic taxonomical concepts of biology, where we can see, incidentally, the existence of a noticeable amount of indecision and indetermination.

Already in the last century, a German biologist, Ernest Haeckele (1834-1919), departing from the Linnaeian concept that makes for two great kingdoms of living things (vegetable and animal) denounced the difficulties of categorising all those microscopic organisms which, because of their characteristics and properties, could not be attributed to either the vegetable or animal kingdom. For these organisms, he proposed a third kingdom, called Protists.
"This vast and complex world includes a range of entities beginning with those that have sub-cellular structure -- existing at the limits of life -- such as viroids and viruses, moving through the mycoplasms, to finally, organisms of greater organisation: bacteria, actinomycetes, mixomycetes, fungi, protozoa, and perhaps even some microscopic algae." (2).
The common element of these organisms is the feeding system, which, being implemented (with very few exceptions) by direct absorption of soluble organic compounds, d ifferentiates them both from animals and vegetables. Animals also feed as above, but especially by ingesting solid organic materials that are then transformed through the digestive process. Vegetables are capable, by utilising mineral compounds and light energy, to feed by synthesising the organic substances.
The contemporary tendency of biologists is to once again pick up, though in a more sophisticated way, the concept of the third kingdom. One goes even further, however, arguing that within that kingdom, fungi must be classified in a distinct category.
O. Verona (3) says that if we put multicellular organisms provided with photosynthetic capabilities (plants) in the first kingdom, and the organisms not provided with photosynthetic pigmentation (animals) in the second kingdom, and organisms from both these kingdoms are made of cells provided with a distinct nucleus (eukaryotes); and, furthermore, if we put in another kingdom (protists) those monocellular organism s that have no chlorophyll and have cells that are without a distinct nucleus (prokaryotes), the fungi can well have their own kingdom because of the absence of photosynthetic pigmentation, the ability to be mono-cellular, and multi-cellular, and, finally, their possession of a distinct nucleus.
Additionally, fungi possess a property that is strange when compared to all other micro-organisms: the ability to have a basic microscopic structure (hypha) with a simultaneous tendency to grow to remarkable dimensions (up to several kilograms), keeping unchanged the capacity to adapt and reproduce at any size.
From this point of view, therefore, fungi cannot be considered true organisms, but cellular aggregatessui generiswith an organismic behaviour, since each cell maintains its survival and reproductive potential intact regardless of the structure in which it exists.
It is therefore clear how difficult it is to identify all the biological processes in such complex l iving realities. In fact, even today, there are huge voids and taxonomical approximations in mycology.
It is worthwhile to examine more deeply this strange world, with such peculiar characteristics, and try to highlight those elements that somehow may be pertinent to the problems of oncology.
1) Fungi are heterotrophic organisms and therefore need, as far as nitrogen and carbon are concerned, pre-formed compounds. Of these compounds, simple carbohydrates, for example monosaccarides (glucose, fructose, and mannose) are among the most utilised sugars. This means that fungi, during their life cycle, depend on other living beings, which must be exploited in different degrees for their feeding. This occurs both in a saprophytic way (that is, by feeding on organic waste) and in a parasitic way (that is, by attacking the tissue of the host directly).
2) Fungi show a great variety of reproductive manifestations (sexual, asexual, gemmation; these manifestations can oft en be observed simultaneously in the same mycete), combined with a great morphostructural variety of organs. All of this is directed toward the end of spore formation, to which the continuity and propagation of the species is entrusted.
3) In mycology, it is often possible to observe a particular phenomenon called heterocarion, characterised by the coexistence of normal and mutant nuclei in cells that have undergone a hyphal fusion.

Nowadays, phitopathologists are quite worried about the creation of individuals that are genetically quite different even from the parents. This difference has taken place by means of those reproductive cycles, which are called parasexual.
The indiscriminate use of phitopharmaceuticals has in fact often determined mutations of the nuclei of many parasite fungi with the consequent creation of heterocarion -- and this is sometimes particularly virulent in its pathogenicity (4).
4) In the parasitic dimension, fungi can develop from the hyphas more or less beak-shaped specialised structures that allow the penetration of the host.
5) The production of spores can be so abundant as to always include, at every cycle, tens, hundreds, and even thousands of millions of elements that can be dispersed at a remarkable distance from the point of origin (a small movement is sufficient, for example, to implement immediate diffusion).
6) Spores have an immense resistance to external aggression, for they are capable of stayingdormantin adverse conditions for many years, while preserving unaltered their regenerative potentialities.
7) The development coefficient of the hyphal apexes after the germination is extremely fast (100 microns per minute under ideal conditions) with ramification capacity, thus with the appearance of a new apex region that in some cases is in the neighbourhood of 40-60 seconds (6).
8) The shape of the fungus is never defined, for it is imposed by the environment in which the fungus develops.
It is possible to observe, for example, the same mycelium in the simple isolated hyphas status in a liquid environment or in the form of aggregates that are increasingly solid and compact up to the formation of pseudoparenchymas and of filaments and mycelial strings (7).
9) By the same token, it is possible to observe in different fungi the same shape whenever they must adapt to the same environment (this is cal led dimorphism).
The partial or total substitution of nourishing substances induces frequent mutations in fungi, and this is further proof of their high adaptability to any sub-strata.
10) When the nutritional conditions are precarious, many fungi react withhyphal fusion(among nearby fungi) which allows them to explore the available material more easily, using more complete physiological processes.
This property, which substitutes co-operation for competition, makes them distinct from any other microorganism, and for this reason Buller calls themsocial organisms(8).
11) When a cell gets old or becomes damaged (i.e. by a toxic substance or by a pharmaceutical) many fungi whose intercellular septums are provided with a pore react by implementing of a defence process calledprotoplasmic fluxthrough which they transfer the nucleus and cytoplasm of the damaged cell into a healthy one, thus conserving unaltered all their biological potential.
12) The phenom ena regulating the development of hyphal ramification are unknown to date (9). They consist either of a rhythmic development, or in the appearance ofsectorswhich, though they originate from the hyphal system, are self-regulating (10), that is, independent of the regulating action and behaviour of the rest of the colony.
13) Fungi are capable of implementing an infinite number of modifications to their own metabolism in order to overcome the defence mechanism of the host. These modifications are implemented through plasmatic and biochemical actions as well as by a volumetric increase (hypertrophy) and numerical hyperplasy of the cells that have been attacked (11).
14) Fungi are so aggressive as to attack not only plants, animal tissue, food supplies and other fungi, but even protozoa, amoebas and nematodes.

Fungi hunt nematodes, for example, with peculiar hyphal modifications that constitute real mycelial criss-cross, viscose, or ring traps that achieve the immobilisation of the worms, as a precursor their hyphal invasion.

In some cases, the aggressive power of fungi is so great as to allow it, with only a cellular ring made up of three units, to tighten in its grip, capture and kill its prey in a short time notwithstanding the prey's desperate struggling.
From the short notations above, therefore, it seems fair to dedicate a greater attention to the world of fungi, especially considering the fact that biologists and microbiologists constantly highlight large deficiencies and voids in all their descriptions and interpretations of the fungi's shape, physiology and reproduction.
So the fungus, which is the most powerful and the most organised micro-organism known, seems to be an extremely logical candidate as a cause of neoplastic proliferation. Imperfect Fungi (so called because of the lack of knowledge and understanding of their biological processes) deserve particular attention since their essential prerogative sits in their fermentative capacity.
The greatest disease of mank ind may therefore hide within the small cluster of pathogenic fungi, and may be after all be located with just some simple deductions able to close the circle and providing the solution.

Considering that, among the human parasite species, the Dermatophytes and Sporotrichum demonstrate an excessively specific morbidity, and that experience shows that Actinomycetes, Toluropsis and Hystoplasma rarely enter the context of pathology, the Candid Albicans clearly emerges as the sole candidate for tumoral proliferation.
If we stop for a second and reflect on its characteristics, we can observe many analogies with neoplastic disease. The most evident are:
1) Ubiquitous attachment: no organ or tissue is spared
2) The constant absence of hyperpyrexia
3) Sporadic and indirect involvement of the differential tissues
4) Invasiveness that is almost exclusively of the focal type
5) Progressive debilitation
6) Refractivity to any type of treatment
7) Proliferation facilitated by multiplicity of indifferent cofounders
8) Symptomatological basic configuration with structure tending to the chronic
Therefore an exceptionally hi gh and diversified pathogenic potentiality exists in this mycete of just a few microns in size, which, even though it cannot be traced with the present experimental instruments, cannot be neglected from the clinical point of view.
Certainly, its present nosological classification cannot be satisfactory, because if we do not keep the possibly endless parasitic configurations in mind, that classification is too simplistic and constraining.

We therefore have to hypothesise that Candida, in the moment it is attacked by the immunological system of the host or by a conventional antimycotic treatment, does not react in the usual, predicted way, but defends itself by transforming itself into ever-smaller and non-differentiated elements that maintain their fecundity intact to the point of hiding their presence both to the host organism and to possible diagnostic investigations.
The Candida's behaviour may be considered to be almost elastic:
When favourable conditions exist, it thrives on an epithelium; as soon as the tissue reaction is engaged, it massively transforms itself into a form that is less productive but impervious to attack -- the spore.

If then continuous sub-epithelial solutions take place coupled with a greater a-reactivity in that very moment the spore gets deeper in the lower connective tissue in such an impervious state, it is irreversible.
In fact, the Candida takes advantage of a structural interchangeability utilising, according to the difficulties to overcome its biological niche.
In this way, Candida is free to expand to maturation in the soil, air, water, vegetation, etc., that is, wherever there is no antibody reaction.
In the epithelium, instead, it takes a mixed form, that is reduced to the sole spore component when it penetrates in the lower epithelial levels, where it tends to expand again in the presence of conditions tissular a-reactivity.

The initial mandatory step of an in-depth research endeavour would be to understandifand in which dimensions the spore transcends; what mechanisms it engages to hide itself or, again, if it preserves its parasitical characteristic, or if it has available a neutral quiescent position, which is difficult or even impossible to detect by the immunological system.
Unfortunately today we do not have the appropriate means, either theoretical or technical, to answer these and similar questions, so that the only valid suggestions can come solely from clinical observation and experience. While not providing immediate solutions, these sources can at stimulate further questions.

Assuming that Candida Albicans is the agent responsible for tumoral development, a targeted therapy would keep into account not just its static and macroscopic manifestations, but even the ultramicroscopic ones especially in their dynamic valence, that is, the reproductive.
It is very probable that the targets to attack are the fungi's dimensional transition points in order to perform a decontamination with such a scope as to include the whole spectrum of the biological expression: parasitic, vegetative, sporal, and even ultra-dimensional and, to the limit, viral.

If we stop at the most evident phenomena, we risk administering salves and unguents forever (in the case of dermatomycosis or in psoriasis), or to clumsily attack (with surgery, radiotherapy or chemotherapy) enigmatic tumoral masses with the sole result of facilitating their propagation, which is already heightened in the mycelial forms.
Why, one may ask, should we assume a different and heightened activity of Candida Albicans since it has been abundantly described in its pathological manifestations?
The answer lies in the fact that it has been studied only in a pathogenic context, that is, only in relation to the epithelial tissues. In reality Candida possesses an aggressive valence that is diversified in function of the target tissue. It is just in the connective or in the connective environment, in fact, and not in the differentiated tissues, that Candida may find conditions favourable to an unlimited expansion.

This emerges if we stop and reflect for a moment on the main function of connective tissue, which is to convey and supply nourishing substances to the cells of the whole organism.
This is to be considered as an environment external to the more differentiated cells such as nervous, muscular, etc. It is in this context, in fact, that the alimentary competition takes place.
On one hand we have the organism's cellular elements trying to defeat all forms of invasion; on the other hand, we have fungal cells trying to absorb ever-growing quantities of nourishing substances, for they have to obey the species' biological imperative to form ever-larger and diffused masses and colonies.

Candida can only exist as saprophyte Second Non-integer epitheliums (erosions, abrasions, etc.), absence of stage debilitating factors, unusual transitory conditions (acidosis, metabolic disorder, and microbial disorder). Candida expands superficially (classic mycosis, both exogenous and endogenous). Third Non-integer epitheliums, presence of debilitating factors (toxic, stage radiant, traumatic, neuropsychic, etc.).

Candida goes deeper into the sub-epithelial levels from which it can be carried to the whole organism through the blood and lymph (intimate mycosis). (12) Stages one and two are the most studied and known, while stage three, though it has been described in its morphological diversity, is reduced to a silent form of saprophytism.

This is not acceptable from a logical point of view, because no one can demonstrate the harmlessness of the fungal cells in the deepest parts of the organism. In fact, the assumption that Candida can behave in the same saprophytic manner that is observed on integer epitheliums when it has successfully penetrated the lower levels is at least risky, because the assumption would have to be sustained by concepts that aretotally aleatory.

In fact, we asked not only to accepta priorithat the connective environment is(a)not suitable to nourish the Candida, but also at the same time to accept(b)the omnipotence of the body's defence system towards an organic structure that is invasive but that then becomes vulnerable once lodged in the deeper tissues.
As to point a), it is difficult to imagine that a micro-organism so able to adapt itself to any sub-strata cannot find elements to support itself in the human organic substance; by the same token, it seems risky to hypothesise that the human organism's defence system is totally efficient at every moment of its existence.
Finally, the assumption that there is a tendency to a state of quiescence and vulnerability in the case of a pathogenic agent such as fungus -- the most invasive and aggressive microorganism existing in nature -- seems to carry a whiff of irresponsible.
It is therefore urgent, on the basis of the above-mentioned considerations, to r ecognise the hazardous nature of such a pathogenic agent, which is capable of easily taking the most various biological configurations, both biochemical and structural, in function of the condition of the host organism.

The fungal expansion gradient in fact becomes steeper as the tissue that is the host of the mycotic invasion becomes less eutrophic, and thus less reactive.
To that end, it seems useful to briefly consider the "benign tumour" nosological entity. This is an issue that always appears in general pathology but that indeed is brushed aside most of the time too easily, and it is overlooked, since it usually doesn't create either problems or worries. It constitutes one of those underestimated grey areas seldom subjected to rational, fresh consideration.

If the benign tumour, however, is not considered a full-fledged tumour, it would be advantageous, for clarity, to categorise it in an appropriate nosological scheme. If, instead, it is thought that it fully belongs to neoplastic pathology, then it is necessary to consider its non-invasive character and consequently to consider the reasons for this.
It is in fact evident how in this second scenario, the thesis based on a presumed predisposition of the organism to auto-phagocytosis, having to admit an expressive graduation, would stumble into such additional difficulties such as to become extremely improbable.
By contrast, in the fungal scenario, the mystery of why there are benign and malignant tumours is exhaustively solved, since they can be recognised as having same etiological genesis.
The benignity or malignancy of a cancer in fact depends on the capability of tissular reaction of a specific organ expressing itself ultimately in the ability to encyst fun gal cells, and to prevent them from developing in ever-larger colonies. This can be achieved more easily where the ratio between differentiated cells and connective tissue is in favour of the former.
Situated between the impervious noble tissues, then, and the defenceless connective, the differentiated connective structures (the glandular structures in particular) represent that medium term which is only somewhat vulnerable to attack, because of an ability to offer a certain type of defence.

And it is in these conditions that benign tumours are formed, that is, where the glandular connective tissue is successful in forming hypertrophic and hyperplastic cellular embankments against the parasites.

In the stomach and in the lung, instead, since there are no specific glandular units, the target organ, provided with a small defensive capability, is at the mercy of the invader. Furthermore, it is worth mentioning how several types of intimate fungal invasion do not determine the appearance of malignant or benign tumours, but a type of particular benign tumour (specific degenerative alterations) as is the case of some organs or apparatuses that do not have peculiar glandular structures, but nevertheless are attacked in their connective tissue, but in a limited way.

If we consider, in fact, multiple sclerosis, SLA, psoriasis, nodular panartherite, etc. the possible development of the fungus in a three-dimensional sense is actually limited by the anatomic configuration of the invaded tissues, so that only a longitudinal expansion is allowed.
Going back to the precondition of a-reactivity that is necessary for neoplastic development in a specific individual, it is permissible to affirm how in the human body each external or internal element that determines a reduction of well-being in an organism, organ or tissue, possesses oncogenic potentiality. This is not so much because of an intrinsic damaging capability as much as for a generic property of favouring the fungal (that is, tumoral) flourishing.
Then the causal network so much invoked in contemporary oncology, which involves toxic, genetic, immunological, psychological, geographical, moral, social, and other factors, finds a correct classification only in a mycotic infectio us perspective where the arithmetical and diachronic summation of harmful elements works as a cofactor to the external aggression.
Having theoretically demonstrated the equivalency tumour = fungus, it is clear how this interpretative key offers a long series of questions concerning the contemporary therapies both oncological (used without reference indexes) and antimycotic (utilised only at a superficial level).
Which path is best to walk today, then, when faced with a cancer patient, since the conventional oncological treatment, not being etiological, can only occasionally have positive effects and most of the time produces damage?

In the fungal perspective in fact, the effectiveness of surgery is noticeably reduced because of the extreme diffusibility and invasiveness characteristic of a mycelial conglomerate. Surgery's to solve the problem is therefore tied to the case -- to conditions, that is, in which one has the luck to completely remove the entire colony (which is often possible in the presence of a sufficient encystment; but here we are in the case of benign tumours).
Chemotherapy and radiotherapy produce almost exclusively negative effects, both for their specific ineffectiveness, and for their high toxicity and harmfulness to the tissues, which in the last analysis favours mycotic aggressiveness.
By contrast, an anti-fungal, anti-tumour specific therapy would keep into account the importance of the connective tissue, together with the reproductive complexity of fungi. Only by attacking the fungi across the spectrum of all its forms, at points where it is most vulnerable from the n utritional point of view, would it be possible to hope to eradicate them from the human organism.

The first step to take, therefore, would be to reinforce the cancer patient with generic reconstituent measures (nutrition, tonics, regulation of rhythms and vital functions), that are able to enhance, by themselves, the general defences of the organism.
Concerning the possibility of having available pharmaceutical cures which unfortunately do not exist today, it seems useful, in the attempt to find an anti-fungal substance that is quite diffusible and therefore effective, to consider the extreme sensitivity of Candida towards sodium bicarbonate (i.e. in the oral candidosis of breasted babies). This is consistent with the fact that Candida has an accentuated ability to reproduce in an acid environment.

Theoretically, therefore, if treatments that put the fungus in direct contact with high bicarbonate concentrations could be found, we should be able to see a regression of the tumoral masses.
And this is what happens in many types of tumour, such as colon, liver -- and especially stomach and lung -- the former susceptible to regression just because of its "external" anatomic position, the latter because of the high diffusibility of sodium bicarbonate in the bronchial system and for its high responsiveness to general reconstituent measures.
By applying a similar therapeutic approach, it has been possible in some patients (about 30 in the last 15 years) to achieve complete remission of the symptomatology and normalisation of the instrumental data.

Following are the reports of seven cases of patients, some of whom survived more than 10 years.
It is important to emphasise that these cases are presented just as an example of what could be a new way of perceiving the complexity of medical problems, especially in oncology.
It is clear, in fact, that because of the very limited number of cases, the lack of documentation showing rigid, orthodox experimental methodology, and the long time that has elapsed since these cases were treated, that the evidence required for strong support of this theory on cancer is lacking.
I will not indicate in this paper the personal, cultural and professional reasons that were responsible for the interruption of the study and cure of cancer patients until recently (that is, until two or three years ago, when I resumed the treatment of cancer cases). I am however convinced that the important fact that some patients have been able to heal and survive for several years with therap ies that are different from the common, deadly therapeutic methods, must be divulged. This is especially because these results come from a new way of thinking which, as opposed to groping in the dark as official and various alternative medicines do, has a well-defined subject -- fungi -- in a theory which of course is still to be proven and validated.
One may ask why more recent cases are not shown below. This is because insufficient time has elapsed since treatment for a demonstration of long-term well being of the patients, and therefore these cases are not included.

It is also important to highlight that nowadays it is very difficult to have a large number of cases, since it is not easy to obtain a large number of cancer patients -- they are addressed by the current system almost exclusively toward the official channels of medicine, even if in many cases those have been proven ineffective or deadly.
Keeping the above in mind, I consider it useful to describe these cases as follows:

Case 1: A 70-year old female patient with diagnosis of stomach adenocarcinoma confirmed by commonly accepted oncological tests (TAK, biopsy, etc.). Two days before the scheduled operation, she accepts the suggestion of trying a less sanguinary approach, and leaves the hospital.
For the period of a month, she is administered sodium bicarbonate (one teaspoon in a glass of water) to ingest half an hour before breakfast (that is, on an empty stomach) for the purpose of maximising the effect.
After about two months normalisation of the gastric function takes place with attenuation at first, and eventual loss of all the symptomatology related to neoplastic pathology (lack of appetite, digestion troubles, fatigue, lipothymic events, etc.).
After an endoscopic examination performed one year after the beginning of therapy, the total remission of neoplastic formation is ascertained and the patient refuses further investigation.
The patient is still alive today, 1 5 years after the treatment.

Case 2: A 67-year-old patient with a long history of gastric ulcer is diagnosed with stomach cancer and a gastrectomy is suggested.
The patient, believing his disease is just an exacerbation of the ulcer, wants to find an alternative to surgery. He therefore accepted a therapy with sodium bicarbonate as in case 1. The therapy determines in a few months the regression of the neoplastic symptomatology.
After about 18 months, during which no check-up is performed, upon the return of symptomatology, treatment is resumed as above. Gastric functionality is quickly re-established and maintained for about eight years, after which contact with the patient is lost.

Case 3: A 58-year-old patient with stomach carcinoma is diagnosed through histological examination performed on endoscopical sample.
The patient chooses not to undergo the conventional therapies and he decides to accept a therapy similar to that in the two preceding cases. The resulting effect is a normalisation of symptomatology for about three years, that is, until there are no further medical check-ups.

Case 4: In September, 1983, a 71-year-old patient undergoes a hospital check-up in a serious condition of emaciation caused by a large weight loss (about 15 Kgs.) which occurred over the prior few months.
Once a stomach neoplastic condition has been diagnosed, and after the layout of a combined oncological therapeutic scheme, the relatives are informed. The relatives are also informed of the difficulties and risks of such treatment, to be administered to such a debilitated patient.
The wife decides to refuse the conventional approach and decides to bring the husband home and try the "harmless" therapy of baking soda, which is administered in a lower dosage than in the preceding cases. That restores appetite and a satisfactory digestive functionality.
For about eight months the patient has difficulty regaining weight. After this, the improvement is more and more evident, with the almost complete regaining of the lost weight (within 24 months) and a considerab le improvement of the patient's general condition.

Case 5: A 51-year-old patient diagnosed at the end of 1983 with bronchial carcinoma in the lower right lobe has the diagnosis confirmed by routine oncological tests (distinctively positive TAK but negative bronchial residue. Surgery is proposed.
The family decides to delay surgery and try the bicarbonate treatment.
Radiological examination is performed 18 months after the treatment. During these months there are no emophtoic episodes as occurred at the beginning of the disease. The radiological examination still indicates the presence of a nodular mass in the lower part of the right lobe, but its dimensions appear to be smaller and the contours of the mass more regular.

Case 6: A 48-year-old patient with tumour in the middle lobe of the lung that has been confirmed by all oncological examinations is put on a waiting list for surgery at the beginning of 1983. Incidentally, the execution modality does not seem to be completely defined because the neoplastic mass exceeds the limits.
The patient leaves the hospital against the advice of doctors - to the point that the doctors look for him for several months. He then submits to a bicarbonate therapy which is able to re-establish healthy conditions.
A radiological examination performed after nine months reveals that the neoplastic mass has been replaced by a tenuous transversal line located at the base of the medium lobe that can be interpreted as a residual scar.
The patient is still living.

Case 7: In 1981, a 55-year-old patient is affected by rectal neoplasy that has been evidentiated through symptoms such as problems with evacuation and bleeding, and, instrumentally, through endoscopic examination. Doctors suggest rectal resection and consequent surgical construction of a preternatural anus. In the attempt to avoid this mutilation, the patient submits to a local therapy with bicarbonate performed with enemas containing a high bicarbonate solution -- 8 teaspoons per litre.
Three years after the treatment, the patient was still living.

Critical considerations

Having explained the theory and having briefly illustrated the cases, it seems appropriate to analyse, in a critical and self-critical spirit, what may emerge in neoplastic pathology that is new and concrete.
If we closely observe the proposed therapeutic approach it is possible to see that, independently of its real effectiveness, it has value as an innovative theory. First, it challenges the present methodology and especially its assumptions. Second, it offers a concrete alternative proposal to a mountain of conjectures and postures that sound authoritative but are too generic and therefore ineffective.
The identification of one tumoral cause, even with all the possible general provisos, would represent a step forward that is indispensable for escaping that passivity determined by a lack of results, and which is responsible for medical behaviours that are based too much on faith and not enough on real confidence.
Given, therefore, that an unconventional me dical approach can benefit some patients better - from any point of view - than the official treatments, and since valuable results can be demonstrated, this should stimulate us to pursue further research while avoiding patronising postures that are both limiting and non-productive.
We can therefore discuss whether or not sodium bicarbonate is the real reason for the recoveries or if, instead, those recoveries are due to the interaction of a number of conditions that have been created, the results of unidentified neuro-psychical factors, or maybe the results of something totally unknown. What is beyond question, however, is the fact that a certain number of people, by not following conventional methods, have been able to go back to normality without suffering and without mutilation.
The message of this experience is therefore a call to search for those solutions that are in accord with the simple Hippocratic premise of man's "well-being"; that is, we must be a stim ulated to a critical evaluation of our contemporary oncological therapies which indubitably can guarantee suffering.

One thing is certain: nowadays it is no longer legitimate (for we are the prey of panic and of the "tumoral syndrome"), to tolerate the slaughtering of patients in the name of a "compassionate" obligation to help and be helped, without the support of solid etiological foundations.
If, for a moment, we take a different point of view and try to look at the world of the tumour with new eyes, that is, by hypothesising a simpler genesis of neoplastic proliferation, even the fungal one, we may be appalled and frightened by the ignorant hand of official medicine - a hand that is armed, however, with great cynicism and profound superficiality.
One could argue that the failures represent the inevitable price to pay to save people's lives. But when the suffering and the "authorised deaths" overwhelm the patient recoveries (that seem, indeed, to be random or due to factors not related to the therapies performed), then it is no longer acceptable to operate at all costs and r egardless of the consequences, for in doing so, we are destined only to hurt people.

One can rebut that the recoveries obtained by using present oncological protocols are not so few, and that in certain types of tumour recoveries are a high percentage. It is easy to see, however, that these results are nothing but the outcome of propaganda sustained by surreptitious argumentation shedding false light on the subject of tumoral nosological entities.
When we group together both malignant tumours that are occasionally or never healed (such as lung and stomach), tumours that border with benignity (such as the majority of thyroid and prostatic tumours, etc.) or put them together with those that have an autonomous positive outcome notwithstanding chemotherapy (i.e. infantile leukaemia) - all of this appears to be devious and misleading, having only the purpose of forging a consensus that would otherwise be impossible to obtain with intellectually ethical behaviour.
If, for example, out of a certain number of tumour species only one is susceptible to reg ression, it is not legitimate to create a nosologic diagram reporting on the global incidence of applied therapeutics regardless of the total neoplasm's. In fact, it would be more appropriate to report the uselessness, even the harmfulness of doing so, and leave an open field for alternative hypotheses as far as the demonstration of positive behaviour by the heteroplasm is concerned.
If, for example, we go back for a moment to infantile leukaemia, the frequent positive outcomes can be correlated with elements that are extraneous to the therapies administered. For example, they can be correlated with those common supportive therapies, which are considered particularly effective in young organisms. They can be correlated with the ability of the connective tissue to acquire, in a particular stage of growth and development, that maturity which is necessary to the strengthening of an immunological activity that is, at a certain point in life, intrinsically insufficient.

It is in fact frequent in medicine that some diseases disappear spontaneously, without apparent reason, but in correlation with certain transitions of organic maturation.
On the oncological-mycological issue, it is known how psoriasis and some chronic and recurrent mycoses of infancy that reject any treatment suddenly, at a certain stage of the body's development, disappear completely without a trace.
From the examples noted, which could be uselessly multiplied ad infinitum, it is evident that the full panorama of tumoral disease is extremely varied and complex. It follows that, taking postures that are exclusive or preclusive, whether they are conventional or unconventional, may indicate a lack of vision. This is especially so since the terrain we are exploring is largely unknown, and therefore cannot be charted in a way that is uniform or standardised.
Wherever we consider an environment occupied by invisible ultra-microscopic elements, and since the struc ture of knowledge must inevitably rest on the construction of a multiplicity of theoretical entities, there is a risk of slipping from a perception that reflects reality to one that is merely fictional. The acceptance of such a fictional construct may become a pernicious reality.
The fact that modern medicine not only cannot offer sufficient interpretative criteria but even uses dangerous methodologies that are also harmful and meaningless - even if carried out with good faith - is something which must push us all to search for humane and logical alternatives. At the same time, it is necessary to carefully, open-mindedly, and logically consider any theory or point of view that is dared to be advanced in the battle against that monstrous and inhuman yoke that is the tumour.

To this end, a note of acknowledgement is to go to all those who are aware of the harmfulness of conventional therapeutic methods and constantly try to find alternative solutions.
People like Di Bella, Govallo and others, though guilty of utilising the same inauspicious principles of official medicine (thus showing an excessively conformist mindset) are actually using common sense by trying to relieve the suffering of cancer patients through the use of painless methodologies and, in some cases, are able to achieve remissions even though in the dark about the real causes of cancer.

In an alternative perspective, then, it would be necessary to conceive a new approach to experimentation in the oncological field, setting epidemiological, etiological, pathogenical, clinical and therapeutical research in line with a renewed microbiology and mycology that would probably drive to the conclusion already illustrated; that is, the tumour is a fungus - the Candida Albicans.
The possible discovery that not only tumours but also the majority of chronic degenerative disease could be reconciled to mycotic causality would represent a qualitative quantum leap, which, by revolutionising medical thinking, could greatly improve life expectancy and quality of life. Such reconciliation might include a wider spectrum of fungal parasites (for example, in diseases of the connective tissues, multiple sclerosis, psoriasis, some epileptic forms, diabetes II, etc.).

In closing, if the world of fungi - those most complex and aggressive micro-organisms - has until now too often been bypassed and left unobserved, the hope of this work is that we will quickly become aware of the hazards of these micro-organisms so that medical resources can be channelled not up blind alleys but toward the real enemies of the human organism: external infectious agents.